British and Chinese researchers have discovered a genetic variant common to Chinese people that make them more susceptible to contracting the swine flu. It was determined that an estimated 25% of Chinese people have this gene variant which is also common to people of Japanese and Korean decent. Tao Dong, lead author of the study and professor at Oxford University, explained: “Understanding why some people may be worse affected than others is crucial in improving our ability to manage flu epidemics and to prevent people dying from the virus.” The researchers studied 83 patients who were diagnosed with swine flu during 2009-2010. Those patients who were part of the study had died from complications from pneumonia, respiratory or kidney failure and it was shown that 69% had this genetic variation.
It was found that 1% of Caucasians have this genetic variant.
The findings of these researchers suggest that those with a genetic predisposition to severe flu should seek out treatment immediately to control the effects and mitigate future possible reactions because they would be susceptible to not only the flu, but also SARS.
It was determined that those with the gene variant (and not limited to those of Chinese descent) would have their immune system compromised which could translate to organ damage, organ failure and blockages of the airways.
Andrew McMichael, co-author of the study and director of the Weatherall Institute of Molecular Medicine at Oxford University, said: “It doesn’t mean you should panic if you have this gene variant. Most people who have it won’t run into any trouble at all.”
McMichael also said: “It remains to be seen how this gene affects the whole picture of influenza in China and South East Asia but it might help explain why new influenza viruses often first appear in this region of the world.”
Michael Osterholm, director of the Center for Infectious Disease Research and Policy at the University of Minnesota, who did not participate in the study, suggests that because an individual contracts an illness does not mean that the effects of that individual is universal and that “it’s the host that does all the damage to themselves.”
Osterholm also believes that genetic screening should be implemented when national flu plans are put into place.
Peter Openshaw, director of the Centre for Respiratory Infection at Imperial College London, believes that: “Further work needs to be done to justify that, but maybe in the future we would be able to say that if you’re of a certain ethnicity, you are more at risk and should be prioritized for vaccination or antivirals. It’s possible we could one day do a genetic test before treating someone with flu to see what the best treatment would be.”
According to the Centers for Disease Control and Prevention (CDC), SARS suddenly appeared between November 2002 and July 2003. An estimated 8,098 people of Asian descent were infected and 774 of them succumbed to the race-specific virus.
SARS, being a respiratory illness, was able to “seed” and spread quite quickly from China to Hong Kong beginning with Carlo Urbani, who discovered the strange virus and later died from it.
In 2003, the World Health Organization (WHO) resolved that they were “deeply concerned that SARS” would be “the first severe infectious disease to emerge in the twenty-first century [and] poses a serious threat to global health security, the livelihood of populations, the functioning of health systems, and the stability and growth of economies.”
Because SARS “requires intensive regional and global collaboration” it was slated to be the next pandemic that would was expected to result in “effective strategies and additional resources at local, national, regional and international levels.”
The original SARS was clearly a genetically created bioweapon.
It had spliced elements of human, cow and mouse viruses which is not possible in nature. Coronaviruses do not jump species – like infecting animals and spreading to humans.
Another tell-tale sign is that the original SARS did not match any known virus. At first officials claimed it was an A virus, then a B virus.
US Army Command and General Staff College at Fort Leavenworth, Kansas published information in a journal intended for military personnel called Ethnic Specific Weapons, wherein the history, desirability and possibilities of biological weapons tailored for specific races of human could be genetically engineered to defend a nation against attack.
Dr. William Schaffner, chairman of preventative medicine at Vanderbilt University, explained: “It took the outbreak in Hong Kong and subsequent spread to bring that to our attention. The surveillance for viruses that cause disease, particularly respiratory viruses, has improved enormously over the last 10 years worldwide. What happened here demonstrates that.”
In 2012, Canadian researchers concluded that the H1N1 flu shot is more dangerous than the flu it purports to protect against. The timing of this study’s release coincided with North American and certain part of Europe preparing to promote massive vaccination efforts on their citizens. Under a propaganda pandemic campaign to fear the public into having the inoculation, this study placed a snag in their plans.
At the Interscience Conference on Antimicrobial Agents and Chemotherapy (ICAAC) study author Dr. Danuta Skowronski explained that in 2008 a study she co-authored proved that the flu shot caused more physiological issues than the H1N1 flu itself.
Skowronski said that “pandemics are infrequent occurrences, but seasonal influenza recurs on an annual basis. It’s a substantial cause of morbidity and mortality and the seasonal vaccine substantially protects against that severe outcome due to seasonal influenza.”
Vaccinations for the H1N1 are ineffective because the inoculation cannot prevent infection or support the body’s development of immunity. However, having been exposed to the flu gives the human body the antibody build-up that facilitates the actions of the human immune system.
Source: Occupy Corporatism
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